Langsung ke konten utama

Pathophysiology of Appendicitis

appendicitis - Pathophysiology of Appendicitis



Appendicitis

The appendix is a small, tube-like organ attached to the first part of the large intestine, also called the colon. It is located in the lower right area of the abdomen. It has no known function. A blockage inside of the appendix causes appendicitis. The blockage leads to increased pressure, problems with blood flow and inflammation. If the blockage is not treated, the appendix can break open and leak infection into the body.


Pathophysiology of Appendicitis

The main cause of appendicitis is obstuksi blockage which can be caused by hyperplasia of the follicles lympoid is the most common cause of fekalit in appendix lumen. The presence of foreign objects such as: worms, stricture due to fibrosis, as a result of previous inflammation. For another example: malignancy (carcinoid carcinoma).

Appendix obstruction that causes mucus produced by mucous unstoppable, more and more mucus is unstoppable and suppress edema appendix wall and stimulate the tunica serosa and visceral peritoneum. Therefore, the neural appendix with the intestinal thorax X then stimulus is perceived as pain around the umbilicus.

The collected mucus was then infected by the bacteria into pus, and then arise venous flow disturbance, while the artery is not disturbed, the resulting widespread inflammation and the local parietal peritoneum, thus causing pain on the right below, this condition is called acute suppurative appendicitis.

When the arterial flow is disrupted then arise allergens and is called by appendicitis gangrenosa. When the wall acute appendix had burst, called appendicitis perforation. When the omentum adjacent bowel can surround the inflamed or perforated appendix will arise a local time, a condition called as appendicitis abscess. In children, because the omentum is still short and thin, relatively long appendix, the appendix wall is much thinner and stamina is still lacking, as well as in older people because there's been blood vessel disorders, the perforation occurred more quickly. If these infiltrates appendicitis cure the symptoms disappear and then arise in the future will occur with chronic appendicitis.
Label:

Komentar

Posting Komentar

Postingan populer dari blog ini

Hematemesis Melena Pathophysiology

A history of dyspepsia, peptic ulcer is the initial allegations. So is a history of recurrent vomiting who initially did not bleed, excessive alcohol consumption leads to the suspicion of gastritis and peptic ulcer disease. A history of recurrent vomiting who initially did not bleed more towards Mallory-Weiss. Excessive alcohol consumption leads to allegations of gastritis (30-40%), peptic ulcer disease (30-40%), or sometimes varicose veins. Weight reduction leads to the suspicion of malignancy. Heavy bleeding accompanied by a clot and refractory shock treatment increases the likelihood of varicose veins. A history of previous abdominal aortic surgery increases the likelihood of fistula aortoenterik. At the young age of patients with a history of upper gastrointestinal bleeding brief repeated (often accompanied by hemodynamic collapse) and a normal endoscopy, should be considered Dieulafoy lesion (a submucosal artery, usually near the heart, which can cause intermittent gastrointestina
Posting Komentar
Baca selengkapnya

Pathophysiology of COPD

COPD, or chronic obstructive pulmonary disease, is a progressive inflammatory disease connecting the airways, lung parenchyma, and vasculature. It causes the damage and remodeling of the airways and lung tissue. Proper functioning of lungs is rejected continuously by COPD. Over a period of time, these changes result in more severe conditions such as pulmonary hypertension and right heart failure. The precise pathophysiology of COPD is unidentified. The inflammatory process is a driving aspect in the pathophysiology of COPD. Recent verification suggests that the inflammatory response results in a number of effects, including an arrival of inflammatory cells such as macrophages, neutrophils and lymphocytes. Thickened airways and structural changes such as increased smooth muscle and fibrosis may also be manifested. Cigarette smoking causes an inflammatory response in the lungs. This response does not cease with the removal of the stimulus, but progresses for an unlimited period of time
Posting Komentar
Baca selengkapnya

Pathophysiology of Typhoid Fever

Typhoid fever , also known as Typhoid , is a common worldwide illness, transmitted by the ingestion of food or water contaminated with the feces of an infected person, which contain the bacterium Salmonella enterica enterica, serovar Typhi. The bacteria then perforate through the intestinal wall and are phagocytosed by macrophages. The organism is a Gram-negative short bacillus that is motile due to its peritrichous flagella. The bacterium grows best at 37°C / 98.6°F – human body temperature. This fever received various names, such as gastric fever, abdominal typhus, infantile remittant fever, slow fever, nervous fever, pythogenic fever, etc. The name of "typhoid" was given by Louis in 1829, as a derivative from typhus . Pathophysiology of Typhoid Fever Transmission of Salmonella typhi can be transmitted through various ways, which is known with 5 M of the Food Fingers, Fomitus (vomiting), Fly, and through Faeces. Faeces and vomiting in patients with typhoid salmonel
Posting Komentar
Baca selengkapnya